Insulin Potentiaton Therapy (IPT)

Evidence Based Data

Insulin Potentiaton Therapy (IPT)

Cancer cells consume more sugar than an average cell and are therefore considered more sensitive to insulin. Insulin is used to potentiate chemotherapy. It does so by attaching itself to the cancerous cells, making them more permeable and thus increasing the toxic effect of chemotherapy on these cells.  This makes it easier to target cancer cells since the excess insulin activators are activated. IPT may also reduce chemotherapy doses, thus reducing the chemo’s side effects.

Insulin Potentiaton Therapy (IPT) Testimonials

How to Kill Cancer Cells with 90% Less Toxicity than Chemo – Dr. Murray Susser, MD
Insulin Potentiation Therapy (IPT Therapy) – Alternative Cancer Treatment
Insulin Potentiation Therapy IPT Alternative Cancer Treatment

Insulin Potentiaton Therapy (IPT)- Relevant Research and News

Low-Dose Chemotherapy with Insulin (IPT) in Combination with Hormone Therapy for Treatment of Castration-Resistant Prostate Cancer

Purpose. To evaluate the results and quality of life of patients with resistant of castration-resistant tumors previously treated with Insulin-potentiation therapy (IPT) combined with hormone therapy.

Materials and methods. Sixteen patients with metastasis prostate tumors after bilateral castration, androgenic blockade, and progression of the disease were observed during the study. The patients were divided into two groups: group A consisting of 8 patients treated with low-dose chemotherapy Epirubicin, Vinblastine, and Cyclophosphamide combined with LHRH agonist and group B consisting of another 8 patients treated with low-dose chemotherapy Docetaxel combined with LHRH agonist.

Results. The overall (groups A and B) results concerning PSA after the sixth IPT show partial effect in 8 out of 16 (50%) patients, stabilization in 4 out of 16 (25%), and progression in 4 out of 16 (25%). The median survival for all treated patients is 11,7 months (range 3–30 months). During the treatment no significant side effects were observed, and no lethal cases occurred.

Conclusion. In spite of the small number of the treated patients with castration-resistant prostate tumors, the preliminary results are promising and this gives us hope and expectations for future serious multicenter research over the possibilities for routine implementation of IPTLD.

Insulin potentiation therapy in the treatment of malignant neoplastic diseases: A three year study

Problem Statement: Even after decades of scientific research, the application of chemotherapy in the
management of neoplastic disease still presents numerous difficulties. Significant, amongst potential complications,
are numerous toxicity-related side effects and the potential for chemoresistance. Despite the widespread tendency
to include a variety of new chemotherapeutics in different combinations, progress in this area has proven slow going
and unsatisfactory due to the aforementioned factors.
Approach: Seeking a new approach, we introduced the method of Insulin Potentiation Therapy (IPT) in our
practice. The theoretical basis and the gathered experimental data on insulin’s mode of action, as well as its
application in practice, show that IPT is a promising method with low toxicity. Moreover, it facilitates a selectively
physiological approach to the management of neoplastic disease using chemotherapy.
In this report we present the results of our three-year experience applying Insulin Potentiation Targeted Therapy
Low Dose (IPTLD) in the treatment of 196 patients diagnosed with a variety of neoplastic diseases.
Results: Our results showed that patients tolerated IPTLD without difficulties, without serious side effects. Our
laboratory tests demonstrated that the dose related toxicity of chemotherapeutics could be largely mitigated when
applied in conjunction with insulin, at a fractionated dose in accordance with a dose dense regimen. Upon follow-up,
eighty-five of 106 patients (80%) with advanced metastatic disease reported a subjectively significant improvement
in their quality of life.
Conclusions: Future extended experimental data and clinical trials would contribute to a more complete
understanding of the therapeutic potential of IPTLD

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