High Dose (IV) Vitamin C for Cancer

Evidence Based Data

High Dose (IV) Vitamin C for Cancer

Over the past century, the notion that vitamin C can be used to treat cancer has generated much controversy. However, new knowledge regarding the pharmacokinetic properties of vitamin C and recent high-profile preclinical studies have revived interest in the utilization of highdose vitamin C for cancer treatment. Studies have shown that pharmacological vitamin C targets many of the mechanisms that cancer cells utilize for their survival and growth.


Cancer cells are fed on sugar during a process that is called glycolysis. Vitamin C blocks glycolysis thus cutting cancer cells’ energy and basically starving the cancerous cells.

Clinics use a high dose of Vitamin C injections usually in conjunction with other treatments.

Scroll down to read relevant scientific research, and find clinics that offer high-dose vitamin C IVs

A Phase II Study of High Dose Vitamin C Intravenous Infusion in Patients with Resectable or Metastatic Solid Tumor Malignancies
Does vitamin C help or hinder cancer patients? | Newshub

Vitamin C IV- Relevant Research and News

Vitamin C Reduce Risk of Breast Cancer Mortality

Vitamin C and survival among women with breast cancer has been studied numerously. In this study, the researchers performed a meta-analysis of 10 studies that were done over the years. A total of 17,696 breast cancer cases were examined, and the results suggest that post-diagnosis vitamin C supplement use may be associated with a reduced risk of mortality. Dietary vitamin C intake was also statistically significantly associated with a reduced risk of total mortality and breast cancer-specific mortality.

Benefits of High-dose Vitamin C on Breast Cancer Cells

a 2019 published article concludes – Combining highdose vitamin C with conventional anti-cancer drugs can have therapeutic advantages against breast cancer cells.

Highdose vitamin C (≥10 mM) significantly decreased cell viability of all breast cancer cell lines, particularly of MCF-7 cells. The catalase activities of MCF7 and MDA-MD-231 cells were also lower than those of MCF10A cells. Moreover, cell viability of both MCF7 and MDA-MD-231 cells was decreased further when combining highdose vitamin C and eribulin mesylate, and this was also true for MCF-7 cells when combining highdose vitamin C with tamoxifen or fulvestrant and for SK-BR3 cells when combining highdose vitamin C with trastuzumab in comparison with chemotherapy or endocrine therapy alone.

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